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Impact of Endocrine Therapy, Menstrual Cycle, PAM50, Ki67 on Treatment Decisions in HR+ and HER2- Breast Cancer

Recruiting
18 years of age
Female
Phase N/A

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Overview

PEAK is a prospective, multicenter, non-interventional investigator-initiated trial (IIT) that aims to investigate the influence of the menstrual cycle phase on Ki67 in patients who either receive Tamoxifen, Aromatase inhibitors ± gonadotropin-releasing hormone (GnRH)-Analogues or nothing or no preoperative endocrine treatment as part of the clinical routine. The investigators moreover address the question whether PAM50 assessment in addition to Ki67 dynamics still impacts treatment recommendations.

Description

Background of the study Chemotherapy and anti-hormonal therapies improve the chances of cure for patients with early-stage hormone receptor (HR)-positive/HER2-negative breast cancer. However, only a few patients benefit from chemotherapy, as classical tumor characteristics such as grading, tumor size, and lymph node involvement in the armpit have a prognostic value but do not allow predictions about the effectiveness of chemotherapy. New treatment concepts, such as gene expression tests and preoperative endocrine therapy, aim to identify patients with a high risk of recurrence and provide them with optimized treatment. Additionally, initial study results indicate that a woman's menstrual cycle could influence the tumor's growth rate. These hints need to be investigated in larger studies to understand how the growth rate of a tumor in different phases of the menstrual cycle can be interpreted.

Purpose of the study The main objective of the PEAK study is to investigate to what extent the menstrual cycle phase influences the tumor growth rate (biomarker Ki67). To make a scientifically sound statement, the growth rate of the tumor in postmenopausal patients must also be examined. In addition, the impact of preoperative anti-hormonal therapy, the dynamics of the growth marker Ki67, and the individual genetic risk (PAM50 gene test) on recommendations for adjuvant therapy in clinical routine should be evaluated. Furthermore, the influence of the aforementioned markers on established clinical-pathological risk factors and the spread of tumor cells should be assessed.

Eligibility

Inclusion Criteria:

  • women ≥ 18 years of age
  • histologically proven unilateral primary non-metastatic invasive breast cancer
  • Estrogen receptor (ER)-/ or Progesterone receptor (PR)- positive and HER2-negative
  • Ki67 from core biopsy is available
  • no lymph-node involvement by clinical evaluation and ultrasound (cN0)
  • not amendable to neoadjuvant chemotherapy
  • surgery or planned surgery at the Department for Women's Health, Tuebingen or Freiburg
  • planned preoperative endocrine treatment with Tamoxifen, Aromatase inhibitors, Goserelin or nothing for 2 - 4 weeks
  • written informed consent

Exclusion Criteria:

  • ER-negative and PR-negative
  • HER2-positive
  • bilateral breast cancer
  • preexisting cancer disease within the last 10 years
  • preexisting invasive ipsi- or contralateral breast cancer (non-invasive ipsi- or contralateral breast cancer is not regarded as an exclusion criteria)
  • any systemic breast cancer therapy before inclusion into the trial
  • indication for neoadjuvant chemotherapy
  • any systemic therapy except Tamoxifen, Aromatase inhibitors, Goserelin before surgery
  • locally advanced, inoperable or metastatic breast cancer
  • pregnant or lactating patients
  • inadequate general condition (not fit for chemotherapy)
  • hormonal contraception within 6 months before inclusion

Study details

Hormone Receptor Positive HER-2 Negative Breast Cancer

NCT05878314

University Hospital Tuebingen

22 June 2024

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