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Modulation of Frontoparietal Dynamics Underlying Adolescent Working Memory Deficits

Modulation of Frontoparietal Dynamics Underlying Adolescent Working Memory Deficits

Non Recruiting
13-25 years
All
Phase N/A

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Overview

The purpose of this study is to test whether a single session of brain stimulation (called repetitive transcranial magnetic stimulation [rTMS]) can improve the brain activity underlying 'working memory.' Working memory is the ability to hold information 'in mind' to complete daily activities. This study involves teenagers with ADHD as well as healthy young adults. It is funded by The COBRE Center for Neuromodulation (CCN) at Butler Hospital (Pilot Project)

Description

The objective of this project is to examine the contributing roles of the PPC and PFC in WM processes and relatedly, develop optimal targets for modulating WM-related neural oscillations. Patient Arm: In a 2x2 factorial double-blind design, we will randomize a sample of adolescents (13-18 years) with WM deficits to intermittent theta burst stimulation (iTBS) at the left dorsolateral prefrontal cortex (DLPFC) or inferior parietal lobule (IPL), based on each participant's structural brain MRI. Control Arm: A sample of healthy young adults (18-25 years) will receive an individualized theta-gamma parameters protocol of iTBS to the left DLPFC. Participants in both arms will complete an active iTBS session and a sham iTBS session. The primary outcome will be theta-gamma coupling during WM demands, as measured via electroencephalography (EEG) during a Sternberg spatial WM task (SWMT) immediately before and after iTBS. The central hypothesis is that the PFC and PPC regions have complimentary roles in executing WM processes. Further, iTBS can modulate theta-gamma coupling in these regions to improve behavioral performance. Our central hypothesis is formulated based on our preliminary data on the critical role of theta-gamma coupling in WM processes. To attain the overall objectives, the following specific aims will be pursued:

Aim 1: Examine the effect of iTBS to the PPC on the encoding stage of WM. Hypothesis: Compared to sham and DLPFC conditions, iTBS to the IPL will increase theta-gamma coupling during encoding. Modulation of theta-gamma coupling will correlate with improved behavioral performance.

Aim 2: Examine the effect of iTBS to the PFC on the maintenance stage of WM. Hypothesis: Compared to sham and IPL conditions, iTBS to the DLPFC will increase theta-gamma coupling during maintenance. Modulation of theta-gamma coupling will correlate with improved behavioral performance.

Aim 3: Examine the feasibility and preliminary effect of individualized iTBS to the PFC. Hypothesis: It will be feasible to utilize peak theta-gamma coupling during the SWMT to identify optimal iTBS parameters. Individualized iTBS will increase theta-gamma coupling during WM demands and improve behavioral performance.

Exploratory Aim: Identify the neocortical circuitry underlying oscillatory modulation. Computational modeling designed for neural interpretation of EEG will translate obtained recordings into cellular/circuit-level activity delineating the neural mechanism of the observed modulation. Hypothesis: pyramidal-interneuron mechanisms within the PPC/PFC will underlie the theta/gamma oscillatory modulation.

Eligibility

Patient Arm: We will enroll a sample of adolescents (age 13-18 years) with working memory

        deficits and ADHD. Participation in this study will not require any adjustments to their
        clinical care. There are no costs to this study (participants compensated) and there are no
        expected long-term benefits to the participants. Participants will be compensated for each
        session. Participants can withdraw from the study at any time.
        Inclusion Criteria
          1. Ability to provide assent and have parent provide parental permission
          2. English fluency of the participant and the legal guardian/parent
          3. 13-18 years
          4. Parent rating on BRIEF-2 Working Memory: Greater than 1.0 SD above normative mean.
          5. IQ > 80
          6. Clinical diagnosis of attention deficit hyperactivity disorder (ADHD): predominantly
             inattentive type, predominantly hyperactive/impulsive type, combined type, or
             unspecified type. Diagnostic criteria will be confirmed with NICHQ Vanderbilt
             Assessment Scales-Parent.
        Healthy Control Arm: We will target a sample of healthy, young adults. Inclusion Criteria
          1. Ability to provide consent
          2. English fluency of the participant
          3. 18-25 years
          4. Never diagnosed or meets current criteria for a psychiatric disorder, as measured by
             self-report of prior diagnoses and formal completion of the diagnostic interview.
        Exclusion Criteria: For patient and healthy control participants
        Participants will be screened to exclude individuals with neurological or medical
        conditions that might confound the results, as well as to exclude participants in whom MRI
        or TMS might result in increased risk of side effects or complications. Common
        contraindications include metallic hardware in the body, cardiac pacemaker, patients with
        an implanted medication pumps or an intracardiac line, or prescription of medications known
        to lower seizure threshold. These account for the majority of the exclusion criteria listed
        below:
          1. Intracranial pathology from a known genetic disorder (e.g., NF1, tuberous sclerosis)
             or from acquired neurologic disease (e.g. stroke, tumor), cerebral palsy, history of
             severe head injury, or significant dysmorphology
          2. History of fainting spells of unknown or undetermined etiology that might constitute
             seizures
          3. History of seizures, diagnosis of epilepsy, or immediate (1st degree relative) family
             history epilepsy
          4. Any progressive (e.g., neurodegenerative) neurological disorder
          5. Chronic (particularly) uncontrolled medical conditions that may cause a medical
             emergency in case of a provoked seizure (cardiac malformation, cardiac dysrhythmia,
             asthma, etc.)
          6. Contraindicated metal implants in the head, brain or spinal cord (excluding dental
             implants, braces or fillings)
          7. Non-removable makeup or piercings
          8. Pacemaker
          9. Implanted medication pump
         10. Vagal nerve stimulator
         11. Deep brain stimulator
         12. TENS unit (unless removed completely for the study)
         13. Ventriculo-peritoneal shunt
         14. Signs of increased intracranial pressure
         15. Intracranial lesion (including incidental finding on MRI)
         16. History of head injury resulting in prolonged loss of consciousness
         17. Substance abuse or dependence within past six months (i.e., DSM-5 substance use
             disorder criteria)
         18. Chronic treatment with prescription medications that decrease cortical seizure
             threshold, not including psychostimulant medication if deemed to be medically safe as
             part of the medical review process.
         19. Active psychosis or mania
         20. Current suicidal intent
         21. Current pregnancy
         22. Significant visual, hearing or speech impairment
         23. Current wards of the state

Study details
    ADHD

NCT05119660

Bradley Hospital

20 August 2025

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